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dc.contributor.authorRios, Austregésilo C.C.-
dc.contributor.authorSalvadori, Daisy M.F.-
dc.contributor.authorOliveira, Susie V.-
dc.contributor.authorRibeiro, Lúcia R.-
dc.creatorRios, Austregésilo C.C.-
dc.creatorSalvadori, Daisy M.F.-
dc.creatorOliveira, Susie V.-
dc.creatorRibeiro, Lúcia R.-
dc.date.accessioned2013-02-20T14:07:00Z-
dc.date.issued1995-
dc.identifier.issn0027-5107-
dc.identifier.urihttp://www.repositorio.ufba.br/ri/handle/ri/8582-
dc.descriptionTexto completo: acesso restrito. p.113–118pt_BR
dc.description.abstractThe genotoxic potential of paraquat was evaluated in mouse somatic (bone marrow) and germ (spermatozoa) cells. Two exposure schedules were conducted: single and multiple treatment. Only for multiple treatment was an increased frequency of chromosomal aberrations observed. In germ cells, paraquat induced an increase of spermshape abnormalities when the animals were treated at three stages of cell development: spermatozoa, spermatid and preleptotene spermatogonial cells. These toxicogenetic effects might be attributed to the ability of paraquat to generate oxygen free radicals.pt_BR
dc.language.isoenpt_BR
dc.sourcehttp://dx.doi.org/10.1016/0027-5107(94)00199-Fpt_BR
dc.subjectParaquatpt_BR
dc.subjectChromosomal aberrationpt_BR
dc.subjectSperm abnormalitypt_BR
dc.titleThe action of the herbicide paraquat on somatic and germ cells of micept_BR
dc.title.alternativeMutation Research - Fundamental and Molecular Mechanisms of Mutagenesispt_BR
dc.typeArtigo de Periódicopt_BR
dc.identifier.numberv. 328, n. 1pt_BR
dc.embargo.liftdate10000-01-01-
Aparece nas coleções:Artigo Publicado em Periódico (EMV)

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